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Protein clues may help explain heart disease risk that persists despite lower LDL cholesterol

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Protein clues may help explain heart disease risk that persists despite lower LDL cholesterol

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According to the World Health Organization (WHO), nearly 17.9 million people die from cardiovascular diseases worldwide every year, with heart attacks and strokes accounting for more than four out of five of those deaths. Coronary heart disease, which can lead to a heart attack if untreated, develops when fatty material (cholesterol) accumulates in the blood vessels supplying the heart. High blood cholesterol is therefore a key risk factor for coronary heart disease.

Lowering LDL cholesterol, often called "bad cholesterol," substantially reduces the risk of heart attacks. Yet some people still develop coronary heart disease even when treatment brings their LDL cholesterol down to a recommended level. Other particles that carry fats through the blood can contribute to this remaining risk, but until now the reason has been unclear.

In a study published in the European Heart Journal, researchers at Chalmers University of Technology, in Sweden, shed new light on the remaining risk of heart disease, even after the known risk factor of bad cholesterol is brought down to a healthy level.

"For this study, we wanted to understand what happens beyond the accumulation of cholesterol in artery walls, whether different fat-carrying particles can damage the blood vessels differently and why the remaining risk of heart disease still occurs," says Jakub Morze, lead author and a postdoctoral fellow at Chalmers University of Technology in Sweden.


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